Search results for "mitochondrial function"

showing 10 items of 14 documents

Arabidopsis RCD1 coordinates chloroplast and mitochondrial functions through interaction with ANAC transcription factors

2019

Reactive oxygen species (ROS)-dependent signaling pathways from chloroplasts and mitochondria merge at the nuclear protein RADICAL-INDUCED CELL DEATH1 (RCD1). RCD1 interacts in vivo and suppresses the activity of the transcription factors ANAC013 and ANAC017, which mediate a ROS-related retrograde signal originating from mitochondrial complex III. Inactivation of RCD1 leads to increased expression of mitochondrial dysfunction stimulon (MDS) genes regulated by ANAC013 and ANAC017. Accumulating MDS gene products, including alternative oxidases (AOXs), affect redox status of the chloroplasts, leading to changes in chloroplast ROS processing and increased protection of photosynthetic apparatus.…

0106 biological sciences0301 basic medicineretrograde signalingChloroplastsArabidopsisPlant BiologyMitochondrion01 natural sciencesElectron Transport Complex IIIGene Expression Regulation PlantArabidopsisOXIDATIVE STRESS-RESPONSETranscriptional regulationCYCLIC ELECTRON FLOWBiology (General)Nuclear proteinANAC transcription factors1183 Plant biology microbiology virologyreactive oxygen speciesbiologyChemistryRETROGRADE REGULATIONGeneral NeuroscienceQRNuclear Proteinsfood and beveragesGeneral MedicinePlants Genetically Modified:Science::Biological sciences [DRNTU]Cell biologyMitochondriaChloroplastviherhiukkasetMedicineSignal transductionmitochondrial functionsResearch ArticleSignal TransductionQH301-705.5SciencemitokondriotGenetics and Molecular BiologyGeneral Biochemistry Genetics and Molecular BiologyPROTEIN COMPLEXESSIGNALING PATHWAYS03 medical and health scienceschloroplastStress PhysiologicalALTERNATIVE OXIDASESkasvitENZYME-ACTIVITIESredox signalingTranscription factorarabidopsis RCD1General Immunology and MicrobiologybiokemiaArabidopsis Proteinsta1182Biology and Life Sciencesbiology.organism_classification030104 developmental biologyCELL-DEATHPLANT-MITOCHONDRIAA. thalianaGeneral BiochemistryRetrograde signalingGENES-ENCODING MITOCHONDRIALproteiinit010606 plant biology & botanyTranscription Factors
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Role of Endoplasmic Reticulum and Oxidative Stress Parameters in the Pathophysiology of Disease-Related Malnutrition in Leukocytes of an Outpatient P…

2019

Cellular pathways such as inflammation or oxidative stress are the cause and triggers of disease-related malnutrition (DRM), but the influence of these markers on endoplasmic reticulum (ER) stress is unknown. The objective of this study was to analyze the relationship between mitochondrial function and ER stress parameters in a DRM population. The study population was composed of 82 outpatient subjects, of whom 45 were diagnosed with DRM and 37 were confirmed to be normonourished according to the American Society for Parenteral and Enteral Nutrition ASPEN criteria. We evaluated anthropometrical and biochemical parameters, pro-inflammatory cytokines in serum. Oxidative and ER stress markers …

AdultMale0301 basic medicinemedicine.medical_specialtyPopulationNutritional Statuslcsh:TX341-641InflammationOxidative phosphorylationEndoplasmic Reticulummedicine.disease_cause03 medical and health sciences0302 clinical medicinemitochondrial functionInternal medicineOutpatientsdisease-related malnutritionLeukocytesmedicineHumans030212 general & internal medicineeducationoutpatient populationAgededucation.field_of_study030109 nutrition & dieteticsNutrition and DieteticsAnthropometryATF6business.industryBrief ReportEndoplasmic reticulumMalnutritionMiddle AgedOxidative StressEndocrinologyinflammationendoplasmic reticulum stressUnfolded protein responseCytokinesFemaleSignal transductionmedicine.symptombusinesslcsh:Nutrition. Foods and food supplyOxidative stressFood ScienceNutrients
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Cytotoxic Potential of the Coelomic Fluid Extracted from the Sea Cucumber Holothuria tubulosa against Triple-Negative MDA-MB231 Breast Cancer Cells

2019

Growing evidence has demonstrated that the extracts of different holothurian species exert beneficial effects on human health. Triple negative breast cancers (TNBC) are highly malignant tumors that present a poor prognosis due to the lack of effective targeted therapies. In the attempt to identify novel compounds that might counteract TNBC cell growth, we studied the effect of the exposure of the TNBC cell line MDA-MB231 to total and filtered aqueous extracts of the coelomic fluid obtained from the sea cucumber Holoturia tubulosa, a widespread species in the Mediterranean Sea. In particular, we examined cell viability and proliferative behaviour, cell cycle distribution, apoptosis, autophag…

0301 basic medicineautophagyCellSettore BIO/05 - ZoologiaBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicinebreast cancermitochondrial functionOrganellemedicineCytotoxic T cellViability assay<i>Holothuria tubulosa</i>Settore BIO/06 - Anatomia Comparata E Citologialcsh:QH301-705.5cell viabilityGeneral Immunology and MicrobiologyHolothuria tubulosaAutophagyCell cyclebiology.organism_classificationHolothuria tubulosa030104 developmental biologymedicine.anatomical_structurelcsh:Biology (General)Apoptosis030220 oncology & carcinogenesisCancer researchcell cycleGeneral Agricultural and Biological Sciencescoelomic fluid
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Does Glycemic Control Modulate the Impairment of NLRP3 Inflammasome Activation in Type 2 Diabetes?

2019

Since mitochondrial dysfunction is associated with NOD-like receptor family protein 3 (NLRP3) activation in type 2 diabetes (T2D), which can eventually lead to an impaired immune response, we set out to determine if glycemic control modulates the effects of T2D on the NLRP3 inflammasome. We have studied leukocytes from 61 diabetic patients [25 with glycated hemoglobin (HbA(1c)) 7% and 36 with HbA(1c) 8%] and 40 healthy controls. Total and mitochondrial reactive oxygen species (ROS) production was enhanced in T2D patients, and mitochondrial ROS was more pronounced in those with poor glycemic control. Levels of gene and protein expression of NLRP3 were decreased in both diabetic groups and mo…

Blood GlucoseMale0301 basic medicineMitochondrial ROSendocrine system diseasesInflammasomesPhysiologyClinical BiochemistryType 2 diabetesmedicine.disease_causeBiochemistrychemistry.chemical_compoundGene expressionoxidative stressGeneral Environmental Scienceintegumentary systemInterleukinInflammasomeMiddle AgedMitochondriaglycaemic controlCytokinesFemaletype 2 diabetesInflammation MediatorsSignal Transductionmedicine.drugmedicine.medical_specialty03 medical and health sciencesmitochondrial functionInternal medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineHumansBody Weights and MeasuresMolecular BiologyAgedGlycemicGlycated Hemoglobin030102 biochemistry & molecular biologybusiness.industrynutritional and metabolic diseasesCell Biologymedicine.diseaseNLRP3 inflammasome030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2chemistryGeneral Earth and Planetary SciencesGlycated hemoglobinReactive Oxygen SpeciesbusinessBiomarkersOxidative stressAntioxidants &amp; Redox Signaling
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Analysis of thiamine transporter genes in sporadic beriberi

2014

Abstract Objective Thiamine or vitamin B 1 deficiency diminishes thiamine-dependent enzymatic activity, alters mitochondrial function, impairs oxidative metabolism, and causes selective neuronal death. We analyzed for the first time, the role of all known mutations within three specific thiamine carrier genes, SLC19 A2, SLC19 A3 , and SLC25 A19 , in a patient with atrophic beriberi, a multiorgan nutritional disease caused by thiamine deficiency. Methods A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema, a subacute sensorimotor neuropathy, and incontinence. Despite normal vitamin B 1 serum levels, his clinical picture was rapidly reverted by high-dose in…

AdultMalemedicine.medical_specialtySLC19 A- SLC25 A19SLC19 AEndocrinology Diabetes and MetabolismGene mutationBeriberimedicine.disease_causeMitochondrial Membrane Transport Proteinslaw.inventionBeriberilawInternal medicineGenotypemedicineThiamine transporterObjective: Thiamine or vitamin B1 deficiency diminishes thiamine-dependent enzymatic activity alters mitochondrial function impairs oxidative metabolism and causes selective neuronal death. We analyzed for the first time the role of all known mutations within three specific thiamine carrier genes SLC19 A2 SLC19 A3 and SLC25 A19 in a patient with atrophic beriberi a multiorgan nutritional disease caused by thiamine deficiency. Methods: A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema a subacute sensorimotor neuropathy and incontinence. Despite normal vitamin B1 serum levels his clinical picture was rapidly reverted by high-dose intramuscular thiamine treatment suggesting a possible genetic resistance. We used polymerase chain reaction followed by amplicon sequencing to study all the known thiamine-related gene mutations identified within the Human Gene Mutation Database. Results: Thirty-seven mutations were tested: 29 in SLC19 A2 6 in SLC19 A3 and 2 in SLC25 A19. Mutational analyses showed a wild-type genotype for all sequences investigated. Conclusion: This is the first genetic study in beriberi disease. We did not detect any known mutation in any of the three genes in a sporadic dry beriberi patient. We cannot exclude a role for other known or unknown mutations in the same genes or in other thiamine-associated genes in the occurrence of this nutritional neuropathy.HumansThiamineGenePolymerase chain reactionGeneticsMutationNutrition and DieteticsbiologyMembrane Transport ProteinsThiamine Deficiencymedicine.diseaseAlcoholismEndocrinologyMutationbiology.proteinThiamineMutations
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Molecular Strategies for Targeting Antioxidants to Mitochondria: Therapeutic Implications

2015

Mitochondrial function and specifically its implication in cellular redox/oxidative balance is fundamental in controlling the life and death of cells, and has been implicated in a wide range of human pathologies. In this context, mitochondrial therapeutics, particularly those involving mitochondria-targeted antioxidants, have attracted increasing interest as potentially effective therapies for several human diseases. For the past 10 years, great progress has been made in the development and functional testing of molecules that specifically target mitochondria, and there has been special focus on compounds with antioxidant properties. In this review, we will discuss several such strategies, …

AntioxidantPhysiologyPlant AlkaloidsCellsAntioxidant propertiesmedicine.medical_treatmentClinical BiochemistryApoptosisContext (language use)Oxidative phosphorylationBiologyMitochondrionBiochemistryCellular redox/oxidative balanceAntioxidantsComprehensive Invited ReviewAutophagymedicineAnimalsHumansRedox activeMolecular BiologyGeneral Environmental ScienceHuman pathologiesAutophagyRedox active moleculesCell BiologyMitochondriaCell biologyBiochemistryGeneral Earth and Planetary SciencesMitochondrial functionTesting of moleculesOxidation-ReductionFunction (biology)Antioxidants &amp; Redox Signaling
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Proteomics Reveals the Potential Protective Mechanism of Hydrogen Sulfide on Retinal Ganglion Cells in an Ischemia/Reperfusion Injury Animal Model

2020

Glaucoma is the leading cause of irreversible blindness and is characterized by progressive retinal ganglion cell (RGC) degeneration. Hydrogen sulfide (H2S) is a potent neurotransmitter and has been proven to protect RGCs against glaucomatous injury in vitro and in vivo. This study is to provide an overall insight of H2S&rsquo

0301 basic medicineneuronal apoptosisgenetic structuresQuantitative proteomicshydrogen sulfidePharmaceutical Sciencelcsh:Medicinelcsh:RS1-441PharmacologyProteomicsRetinal ganglionArticlelabel-free mass spectrometrylcsh:Pharmacy and materia medica03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemitochondrial functionIn vivoDrug DiscoverymedicineRetinaChemistrylcsh:RRetinalmedicine.diseaseequipment and supplieseye diseases030104 developmental biologymedicine.anatomical_structureglaucomaRetinal ganglion cellMolecular Medicinesense organsReperfusion injurysignalling pathways030217 neurology & neurosurgeryPharmaceuticals
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L-karnitīna biosintēzes inhibitora mildronāta neirotropo efektu pētījumi eksperimentālajos modeļos

2014

Elektroniskā versija nesatur pielikumus

cerebrālā išēmijaL-karnitīnsmildronatePharmaceutical pharmacologysexual behaviourseksuālā uzvedībamitohondriju funkcijastrokemitochondrial functionmildronātsFarmaceitiskā farmakoloģijaL-carnitineMedicineMedicīna un farmācijaVeselības aprūpeMedicīna
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Alteration of the Mitochondrial Effects of Ceria Nanoparticles by Gold: An Approach for the Mitochondrial Modulation of Cells Based on Nanomedicine

2020

Ceria nanoparticles are cell compatible antioxidants whose activity can be enhanced by gold deposition and by surface functionalization with positive triphenylphosphonium units to selectively target the mitochondria. The antioxidant properties of these nanoparticles can serve as the basis of a new strategy for the treatment of several disorders exhibiting oxidative stress, such as cancer, diabetes or Alzheimer&rsquo

AntioxidantantioxidantGeneral Chemical Engineeringmedicine.medical_treatmentNanoparticleceria nanoparticles02 engineering and technologyMitochondrionmedicine.disease_causeArticlelcsh:Chemistry03 medical and health scienceschemistry.chemical_compoundQUIMICA ORGANICAmitochondrial functionmedicineGeneral Materials ScienceNRF1Gold-supported ceria nanoparticles030304 developmental biology0303 health sciencesChemistryfungigold-supported ceria nanoparticlesfood and beveragestriphenylphosphonium gold-supported ceria nanoparticles021001 nanoscience & nanotechnologylcsh:QD1-999Colloidal goldBiophysicsNanomedicineMitochondrial functionAntioxidant0210 nano-technologyAdenosine triphosphateCeria nanoparticlesOxidative stressTriphenylphosphonium gold-supported ceria nanoparticles
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Mitochondrial dysfunction in cholestatic liver diseases

2011

et al.

medicine.medical_specialtyMitochondrial DNABiliary cirrhosisMitochondrial HepatopathyApoptosisReviewBiologyMitochondrionmedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyLiver diseaseCholestasisInternal medicinemedicineHumansBiología y BiomedicinaCholestasisGeneral Immunology and MicrobiologyLiver Diseasesmedicine.diseaseBile acidsCell biologyMitochondriaEndocrinologyMitochondrial biogenesisOxidative stressMitochondrial functionMitochondrial dysfunctionOxidative stressFrontiers in Bioscience (Elite edition) 4: 2233-2252 (2012)
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